Timur S. Engin is a partner in the law firm of McDermott Will & Emery LLP and is based in the Firm's Silicon Valley office. He is a member of the Intellectual Property, Media & Technology Department. Timur focuses his practice on patent litigation with experience in the biotechnology, semiconductor, computer networking, geographic information system, wireless communications, medical device, chemical, consumer appliance, commercial graphics and digital video recorder industries.
Prior to joining McDermott, Timur practiced patent litigation in the Silicon Valley office of a global law firm. He has prosecuted patents related to medical devices and techniques, environmental biology and computer networking. Timur has also represented individual and corporate clients in private securities litigation and is experienced in internal corporate investigations.
Timur earned his J.D. from the University of Virginia School of Law. At Virginia, he was a William Minor Lile Moot Court semifinalist and a member of the Virginia Tax Review editorial board. Timur earned a B.S. in biology and a minor in chemistry from the University of South Carolina, Honors College, where he was a member of Phi Beta Kappa.
The Board of Governors of the State Bar of California has twice presented Timur with the Wiley W. Manuel Award for Pro Bono Legal Services for his volunteer legal work for indigent clients.
Timur is registered to practice before the U.S. Patent and Trademark Office and is admitted to the bars of California and Massachusetts. He is also admitted to practice in the U.S. District Court for the District of Massachusetts, the Northern District of California and the Court of Appeals for the Federal Circuit.
As an undergraduate, Timur performed molecular biological research in the Department of Pathology at the USC School of Medicine. His publications include:
- Akerman, G.S., Tolleson, W.H., Brown, K.L., Zyzak, L.L., Mourateva, E., Engin, T.S.,Basaraba, A., Coker, A.L., Creek, K.E. and Pirisi, L. Human papillomavirus type 16 E6 and E7 cooperate to increase epidermal growth factor receptor (EGFR) mRNA levels, overcoming mechanisms by which excessive EGFR signaling shortens the life span of normal human keratinocytes. Cancer Res. 61:3837-3843, 2001.
- Borger, D.R., Mi, Y., Geslani, G., Zyzak, L.L., Batova, A., Engin, T.S., Pirisi, L. and Creek, K.E. Retinoic acid resistance at late stages of human papillomavirus type 16- mediated transformation of human keratinocytes arises despite intact retinoid signaling and is due to a loss of sensitivity to transforming growth factor-beta. Virology 270:397-407, 2000.
Representative Experience
Some recent representative matters include:
- Representing a leading wireless communications company in a patent infringement action pending before the International Trade Commission.
- Represented a public university asserting a pioneering biotechnology patent against a major agricultural company. The case settled for in excess of $200 million the day before trial.
Education
- University of Virginia School of Law, J.D., 2000
- University of South Carolina Honors College, B.S. (magna cum laude), 1997
